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Antifungal drug resistance evoked through RNAi-dependent epimutations Long term mutations were once regarded as the only way for drug-resistant strains to evolve Microorganisms like bacteria and fungi can evade treatment by acquiring mutations in the genes targeted by antibiotics or antifungal drugs. These permanent mutations were once regarded as the only method for drug-resistant strains to evolve. Now a new study shows that microorganisms may use a temporary silencing of medication targets – – known as epimutations – – to gain the benefits of drug level of resistance without the commitment. Although new mechanism was uncovered in a fungus known as Mucor circinelloides, chances are to be used by other fungi along with bacteria, viruses and other organisms to withstand treatment with numerous drugs.